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Light and shadow. Covered with an unusual shadow. A further wave of lighter-skinned populations into Europe and elsewhere is associated with the Yamnaya culture and the Indo-European migrations.

The modern association between skin tone and latitude is thus a relatively recent development. It is widely supposed that light skin pigmentation developed due to the importance of maintaining vitamin D3 production in the skin.

In the s, biochemist W. The major positive effect of UV radiation in land-living vertebrates is the ability to synthesize vitamin D3 from it. A certain amount of vitamin D helps the body to absorb more calcium which is essential for building and maintaining bones, especially for developing embryos.

Vitamin D production depends on exposure to sunlight. Humans living at latitudes far from the equator developed light skin in order to help absorb more vitamin D.

People with light type II skin can produce previtamin D3 in their skin at rates 5—10 times faster than dark-skinned type V people.

In , anthropologist Nina Jablonski and her husband George Chaplin collected spectrometer data to measure UV radiation levels around the world and compared it to published information on the skin color of indigenous populations of more 50 countries.

Jablonski points out that people living above the latitudes of 50 degrees have the highest chance of developing vitamin D deficiency.

She suggests that people living far from the equator developed light skin to produce adequate amounts of vitamin D during winter with low levels of UV radiation.

Genetic studies suggest that light-skinned humans have been selected for multiple times. These regions were uninhabited by humans until about 12, years ago.

In northern Fennoscandia at least, human populations arrived soon after deglaciation. Reindeer meat, organs, and fat contain large amounts of vitamin D which the reindeer get from eating substantial amounts of lichen.

As their founding populations lacked alleles for light skin colour, they may have had insufficient time for significantly lower melanin production to have been selected for by nature.

Two other main hypotheses have been put forward to explain the development of light skin pigmentation: resistance to cold injury, and genetic drift; now both of them are considered unlikely to be the main mechanism behind the evolution of light skin.

The resistance to cold injury hypothesis claimed that dark skin was selected against in cold climates far from the equator and in higher altitudes as dark skin was more affected by frostbite.

The supposition that dark skin evolved in the absence of selective pressure was put forward by the probable mutation effect hypothesis.

The subsequent spread of light skin was thought to be caused by assortive mating [36] and sexual selection contributed to an even lighter pigmentation in females.

Increasingly lighter skinned populations are distributed across areas with incrementally lower levels of UV radiation. Melanin is a derivative of the amino acid tyrosine.

Eumelanin is the dominant form of melanin found in human skin. Melanin is produced in specialized cells called melanocytes , which are found in the lowest level of the epidermis.

Humans with naturally occurring light skin have varied amounts of smaller and scarcely distributed eumelanin and its lighter-coloured relative, pheomelanin.

For the same body region, individuals, independently of skin colour, have the same amount of melanocytes however variation between different body parts is substantial , but organelles which contain pigments, called melanosomes, are smaller and less numerous in light-skinned humans.

For people with very light skin, the skin gets most of its colour from the bluish-white connective tissue in the dermis and from the haemoglobin associated blood cells circulating in the capillaries of the dermis.

The colour associated with the circulating haemoglobin become more obvious, especially in the face, when arterioles dilate and become tumefied with blood as a result of prolonged physical exercise or stimulation of the sympathetic nervous system usually embarrassment or anger.

The characteristic of fair skin, red hair , and freckling is associated with high amount of pheomelanin, little amounts of eumelanin. This phenotype is caused by a loss-of-function mutation in the melanocortin 1 receptor MC1R gene.

Freckles usually only occur in people with very lightly pigmented skin. They vary from very dark to brown in colour and develop a random pattern on the skin of the individual.

This can contribute to premature aging and skin cancer. Normal tanning is usually delayed as it takes time for the melanins to move up in the epidermis.

Heavy tanning does not approach the photoprotective effect against UVR-induced DNA damage compared to naturally occurring dark skin , [63] [64] however it offers great protection against seasonal variations in UVR.

Gradually developed tan in the spring prevents sunburns in the summer. This mechanism is almost certainly the evolutionary reason behind the development of tanning behaviour.

Skin pigmentation is an evolutionary adaptation to the various UV radiation levels around the world.

There are health implications of light-skinned people living in environments of high UV radiation. Various cultural practices increase problems related to health conditions of light skin, for example sunbathing among the light-skinned.

Humans with light skin pigmentation living in low sunlight environments experience increased vitamin D synthesis compared to humans with dark skin pigmentation due to the ability to absorb more sunlight.

Almost every part of the human body, including the skeleton, the immune system, and brain requires vitamin D. Sunlight is necessary for the production of vitamin D.

Vitamin D production in the skin begins when UV radiation penetrates the skin and interacts with a cholesterol-like molecule produce pre-vitamin D3.

The farther a place is from the equator, the less UVB is received, and the potential to produce of vitamin D is diminished.

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